The treatment of the human form of mad cow disease could be revolutionised by scientists at the University of Liverpool. They have determined the atomic structure of the binding between a brain protein and an antibody that could treat variant Creutzfeldt-Jakob disease (vCJD). vCJD is part of a family of neurodegenerative disorders known as prion diseases, which can develop when a naturally occurring protein called PrP comes into contact with infected prions. Antibodies that "stick" to PrP could treat vCJD.
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